RUOResearch Use Only · Not FDA-cleared

GLP-1 &
Metabolic Response

The most comprehensive GLP-1 genetic report available. 9 genes, 20+ variants, 3 clinical dimensions -- will it work, why you eat the way you do, and which drug fits your biology. Includes two exclusive pathways found in no other consumer GLP-1 test: leptin resistance (LEP/LEPR) and drug interaction watch (CYP2D6).

⚠ Research Use Only. Not FDA-cleared or approved. Not intended to diagnose, treat, cure, or prevent any disease. Always discuss findings with a licensed healthcare provider.

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Key variants

RSIDs analyzed in this panel

Representative variants. Full report includes all associated loci with genotyped values, effect sizes, and clinical interpretation.

rs6923761
GLP1R Gly168Ser · semaglutide response
Determines receptor binding efficiency for semaglutide and related drugs (p=6.0x10-⁵). Your report shows your genotype and personalized response tier.
rs1800437
GIPR Glu354Gln · tirzepatide tolerability
Modulates GI side-effect risk on tirzepatide's dual GLP-1/GIP mechanism. Your report shows your genotype and personalized risk tier. (Nature 2026 GWAS, N=27,885)
rs9939609
FTO · brain satiety & reward circuit
fMRI-validated satiety signaling variant, the strongest common GLP-1 response candidate. Your report shows your genotype and personalized profile.
rs17782313
MC4R · hypothalamic appetite + CV lipid risk
Affects appetite regulation and cardiovascular lipid risk independent of body weight. Your report shows your genotype and personalized profile.
rs1137101
LEPR Gln223Arg · leptin receptor sensitivity
Affects leptin receptor sensitivity, a driver of GLP-1 response ceiling. Your report shows your genotype and personalized profile.
rs3892097
CYP2D6*4 · drug interaction watch
Generates a personalized drug-interaction watch list for medications co-prescribed with GLP-1 therapy. Your report shows your genotype and guidance.
Evidence base

Supporting literature

Phan A. et al. (2025) Obesity -- semaglutide 2.4mg n=112, GLP1R rs6923761 sex-modified response. Nature 2026 GWAS (N=27,885) -- first genome-wide GLP-1 RA predictors; GIPR rs1800437 tirzepatide-specific signal. Dawed et al. Lancet Diabetes & Endocrinology 2023 (n=4,571 across 5 RCTs). Godschall et al. Nature 2026 -- GLP-1 central amygdala reward circuit mechanism. Zorn et al. Nature Medicine 2025 -- MC4R cardiovascular lipid independence (n=7,719). Hu et al. Endocr Connect 2025 -- leptin resistance review. Jayathilaka et al. Clin Pharmacol Drug Dev 2025 -- ARRB1 & CYP2D6 indirect interaction.

Layer 1: Will It Work?

GLP1R encodes the receptor that semaglutide and all GLP-1 drugs bind to. Carriers of rs6923761 show 1.64%/month weight loss vs. 1.04% in non-carriers in a prospective semaglutide study -- and 0% of women with the AA genotype were non-responders. ARRB1 controls receptor internalization and biased agonism, explaining why some patients plateau earlier and why dose escalation helps some but not others. These two genes determine the fundamental biological ceiling of GLP-1 efficacy for you specifically.

Layer 2: Why You Eat the Way You Do

FTO rs9939609 AA carriers consume ~350 more kcal after a standardized meal because their brains keep responding to food cues even after eating -- confirmed by fMRI. A 2026 Nature paper confirmed GLP-1 drugs suppress exactly this neural circuit via the central amygdala. MC4R rs17782313 variants affect not just appetite but cardiovascular lipid risk independently of body weight (7,719-person Nature Medicine 2025 study). LEP/LEPR variants drive leptin resistance -- the silent background factor explaining why some patients need higher doses or combination approaches. AuraGen is the only GLP-1 test that includes this pathway.

Layer 3: Which Drug, What Risk

GIPR rs1800437 determines how much benefit you get from tirzepatide's dual GLP-1 + GIP mechanism -- and whether the GIP arm elevates vomiting risk (15x odds when combined with GLP1R effect allele, Nature 2026). CTRB1 rs7202877 is included as a tolerability and incretin-pathway flag with full transparency: it has strong DPP-4 inhibitor evidence but GLP-1 RA efficacy is non-significant (OR 1.12, p=0.84). CYP2D6 status generates a personalized drug interaction watch list for commonly co-prescribed antidepressants and beta-blockers whose absorption is altered by GLP-1's effect on gastric emptying -- a safety feature found in no other GLP-1 genetic report.

How to read this report

Every variant row shows two badges: a genotype status badge (CARRIER / RISK ALLELE / REFERENCE / NOT DETECTED) and an evidence level badge (Level A = strong, multiple studies or CPIC/FDA guideline; Level B = moderate, single study or mechanistic). AuraGen is transparent where evidence is strong vs. emerging -- we never present all variants as equivalent predictors. This is the only GLP-1 genetic test that discloses evidence grades variant-by-variant.

⚠ Research Use Only (RUO)

This report is for Research Use Only and is not FDA-cleared, FDA-approved, or CE-IVD certified. Polygenic risk scores and pharmacogenomic findings reflect statistical associations from published peer-reviewed GWAS studies. Results are not diagnostic and do not constitute medical advice. Always consult a licensed physician, genetic counselor, or pharmacist before acting on any finding. © 2026 AuraGen Wellness · auragenwellness.com

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  • Layer 1 -- Will It Work? GLP1R (4 variants) + ARRB1 -- direct receptor response genes
  • Layer 2 -- Why You Eat: FTO rs9939609 (fMRI-validated satiety), MC4R rs17782313 (CV module), CNR1 (cravings), LEP/LEPR (leptin resistance) -- EXCLUSIVE
  • Layer 3 -- Drug Match & Risk: GIPR rs1800437 (tirzepatide-specific), CTRB1 (tolerability), CYP2D6 (drug interaction watch) -- EXCLUSIVE
  • Drug match module: semaglutide vs. tirzepatide vs. retatrutide recommendation based on your genotype
  • Interpretation key: Level A / Level B evidence grading with full transparency disclosures
  • Powered by Nature 2026 GWAS (N=27,885) + Lancet Diabetes & Endocrinology 2023 (n=4,571) + 7 peer-reviewed citations
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⚠ Research Use Only · Not FDA cleared